Acidum aceticum paints the pale, passive collapse of the cachectic organism: waxy transparency of the skin, cold clammy sweat, unquenchable thirst for cold water (in small, repeated sips), emaciation despite food, and a proneness to passive bright-red hæmorrhage and dropsical effusions. The blood is thinned, the vessels ooze, the serous cavities fill, and the stomach burns and sours; meanwhile the kidneys waste water (often sugar), yet strength ebbs after every evacuation—stool, sweat, urine, or bleed. At the bedside, Acet-ac. is not demonstrative: no feverish bustle, no frantic restlessness. Instead there is a quiet sinking—a patient who lies still, pale and perspiring, asks for cold sips, and faints with trifling losses or exertions. This passivity contrasts with Arsenicum (burning anxiety, hot sips) and Veratrum (agonised spasms and violent purging).
The remedy’s axes interlock: Blood (anæmia/bleeding) ↔ Stomach (burning/sour vomiting) ↔ Kidney (polyuria/diabetes) ↔ Serosa (dropsy). The modalities clinch the choice: worse night, worse from loss of fluids, worse from milk/fats and large draughts; better for rest, cool air, small cold sips, gentle pressure/warmth to the epigastrium. In hæmorrhage Acet-ac. suits the bright-red passive bleeder who turns waxy and faint from a little loss; in choleraic states the sour, watery vomiting, rice-water stools, cold sweat, and quiet collapse are characteristic; in diabetes the pale, copious urine, great thirst, night sweats, and emaciation point the way. When dropsy supervenes—ankles pitting, abdomen tense, chest oppressed—the same thirst-sweat-pallor triad persists. Across these theatres the prescriber should hear Acet-ac.’s refrain: cold sips, waxy sweat, passive oozing, and quiet sinking—a signature not to be overlooked in small, stubborn, wasting cases. [Hering], [Allen], [Clarke], [Boericke], [Boger], [Nash], [Phatak], [Hughes], [Kent].
